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Creators/Authors contains: "Rodriguez, Facundo"

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  1. ABSTRACT We use the improved IllustrisTNG300 magnetohydrodynamical cosmological simulation to revisit the effect that secondary halo bias has on the clustering of the central galaxy population. With a side length of 205 h−1 Mpc and significant improvements on the subgrid model with respect to previous Illustris simulations, IllustrisTNG300 allows us to explore the dependencies of galaxy clustering over a large cosmological volume and halo mass range. We show at high statistical significance that the halo assembly bias signal (i.e. the secondary dependence of halo bias on halo formation redshift) manifests itself on the clustering of the galaxy population when this is split by stellar mass, colour, specific star formation rate, and surface density. A significant signal is also found for galaxy size: at fixed halo mass, larger galaxies are more tightly clustered than smaller galaxies. This effect, in contrast to the rest of the dependencies, seems to be uncorrelated with halo formation time, with some small correlation only detected for halo spin. We also explore the transmission of the spin bias signal, i.e. the secondary dependence of halo bias on halo spin. Although galaxy spin retains little information about the total halo spin, the correlation is enough to produce a significant galaxy spin bias signal. We discuss possible ways to probe this effect with observations. 
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  2. null (Ed.)
    The approximately 100 billion neurons in our brain are responsible for everything we do and experience. Experiments aimed at discovering how these cells encode and process information generate vast amounts of data. These data span multiple scales, from interactions between individual molecules to coordinated waves of electrical activity that spread across the entire brain surface. To understand how the brain works, we must combine and make sense of these diverse types of information. Computational modeling provides one way of doing this. Using equations, we can calculate the chemical and electrical changes that take place in neurons. We can then build models of neurons and neural circuits that reproduce the patterns of activity seen in experiments. Exploring these models can provide insights into how the brain itself works. Several software tools are available to simulate neural circuits, but none provide an easy way of incorporating data that span different scales, from molecules to cells to networks. Moreover, most of the models require familiarity with computer programming. Dura-Bernal et al. have now developed a new software tool called NetPyNE, which allows users without programming expertise to build sophisticated models of brain circuits. It features a user-friendly interface for defining the properties of the model at molecular, cellular and circuit scales. It also provides an easy and automated method to identify the properties of the model that enable it to reproduce experimental data. Finally, NetPyNE makes it possible to run the model on supercomputers and offers a variety of ways to visualize and analyze the resulting output. Users can save the model and output in standardized formats, making them accessible to as many people as possible. Researchers in labs across the world have used NetPyNE to study different brain regions, phenomena and diseases. The software also features in courses that introduce students to neurobiology and computational modeling. NetPyNE can help to interpret isolated experimental findings, and also makes it easier to explore interactions between brain activity at different scales. This will enable researchers to decipher how the brain encodes and processes information, and ultimately could make it easier to understand and treat brain disorders. 
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